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QuackWatch.com Rebuttal
When you take on the A.M.A. and the various Pharmaceutical Giants, as has Dr. Wallach, you are bound to make some enemies. One website, in particular, www.quackwatch.com/01QuackeryRelatedTopics/DSH/colloidalminerals , is especially vociferous in its denouncement of Dr.Wallach and all he stands for. To set the record straight, we include the following rebuttal to the various charges and complaints contained on QuackWatch, courtesy of Professor emeritus, Dr. G. N. Schrauser. Investigating
Dr. J.D. Wallach’s Claims. By G.N. Schrauzer[1] Dr. Joel Wallach is a veterinarian, naturopathic physician,
author and a popular lecturer. He
obtained his degree of Doctor of Veterinary Medicine (D.V.M.) in 1964 and a B.S.
degree in agriculture in 1962 (Major animal husbandry) from the University of
Missouri. From 1966 to 1967 he held
a post doctoral fellowship in comparative medicine at the Center for the Biology
of Natural Systems, George Washington University, St. Louis.
Thereafter he held positions at Iowa State University Diagnostic
Laboratory, Ames, Iowa, and Natal Fish & Game Department, Natal Republic of
South Africa and subsequently joined the St. Louis Zoological Gardens as a
Wildlife Veterinarian and pathologist. This
provided him with the unique opportunity to observe, diagnose and treat a great
number of captive wild animals. In
addition he performed autopsies on a large number of animals dying in zoos in
St. Louis, Chicago, Los Angeles, Jacksonville and Memphis.
Later, at Yerkes Regional Primate Research Center, Department of
Pathology, Atlanta, Georgia, Wallach conducted comparative autoptic studies on
primates as well as on humans. In
1979, he lost his position at Yerkes after he proposed, on the basis of
observations on rhesus monkeys, that cystic fibrosis in humans was an acquired
and treatable condition rather than congenital disorder.
In 1980, Dr. Wallach joined the Faculty of the National College of
Naturopathic Medicine in Portland, Oregon, where he taught in the area of
nutrition while pursuing an N.D. degree in 1982.
After obtaining his N.D. degree and license in 1982, Wallach went into
private practice in Cannon Beach, Oregon, specializing in the nutritional
treatment of cystic fibrosis patients. To
continue his research on cystic fibrosis in 1987, he traveled to China with his
wife, Dr. Ma Lan, a Chinese physician, and conducted a study at Harbin Medical
University. From 1990 to 1993,
Wallach worked as a naturopathic physician for Hospital Santa Monica in Tijuana,
thereafter he entered the multilevel marketing business and in 1997 founded his
own company, American Longevity. Wallach
played a major role in the development of the market of liquid vitamin –
mineral supplements. He became
nationally known through his widely distributed audiotape, “Dead Doctors
Don’t Lie,” wherein, in essence, he proposes that humans can achieve their
maximum biological life span through proper nutrition and an adequate supply of
vitamins and minerals. To attain a
long life, he advises people to take charge of their own health rather than rely
on the advice of their physicians, who, in his view, make poor role models in
terms of their own health and longevity. Wallach
is a dynamic, humorous speaker who fearlessly attacks the weaknesses of our
present health care system. This
rendered him popular among his adherents, but also earned him the scorn of
critics and the medical establishment, who periodically express their
displeasure about his opinions and persona in magazine articles of the
electronic media. The present
account analyzes some of his statements and views which are most often cited in
attempts to discredit him. Mineral depleted food crops – fact or illusion? In his audiotape “Dead Doctors Don’t Lie,” Wallach
claims that depleted soils give rise to mineral deficiency diseases which
threaten the health of Americans. In
support he cites U.S. Senate Document 264, of 1936, which states: Erosion and unwise farming methods have led to mineral depleted soils resulting in mineral deficient plants, livestock and people…The alarming fact is that food now being raised on millions of acres of land that no longer contain enough of certain minerals are starving us – no matter of how much of them we eat…Laboratory tests prove that the fruit, vegetables, grains, eggs and even the milk and meats of today are not what they were a few generations ago…It is bad news to learn from our leading authorities that 99% of the American people are efficient in these minerals” [74th Congress, 2nd Session, 1036]. One of
Wallach’s critics recently labeled U.S. Senate Document 264 a “baseless
opinion piece” and also rejected
the notion mineral deficiencies adversely affect the health and longevity of
Americans.1
That anyone would say this today must astonish, since it has been known
for quite some time that our agricultural soils in many areas lack certain
minerals. One of the first elements
found to be lacking in close to two thirds of the arable land was iodine, and
these areas were known as the “Goiter Belt.”
Iodine deficiency not only severly affected the health of humans; in
Montana alone, 1 million pigs died annually from iodine deficiency before
iodized fodders were introduced. Later,
areas deficient in cobalt, copper, phosphorus, manganese, zinc, boron, fluorine
and selenium were identified. 2,3
Food crops from soils low in selenium not only causes animals to develop
fatal selenium deficiency diseases, human health
is also adversely affected, as evidenced by studies which revealed that human
cancer mortalities are higher in low selenium regions.
The need for additional selenium in our food chain thus became apparent;
a recently concluded 10 year trial 4
showed that human cancer risk can be significantly reduced by supplemental
selenium. The view thus can be
defended that many Americans are dying prematurely of cancer because they are
not getting enough of a needed
essential mineral, selenium, with their food. That not all may be right with our food crops was also concluded by the authors of the well documented, 735 page Kellogg Report 5 of 1989. This report, which addresses the impact of nutrition, environment and lifestyle on the health of Americans, warns that Wallach thus is not the only to warn about the consequences of soil depletion and other aspects of intensive agriculture. The Longevity of physicians – who is right? Wallach claims that American medical doctors have an
average lifespan of only 58.5 years and hence are poor role models of longevity.
His critics argue that doctors actually have a greater life expectancy
than the general population. 1
Wallach calculated the average age at death from the obituary listings in
the Journal of the American Medical Association.
For the years Wallach quotes this result is indeed obtained.
Calculations with a larger database show that doctors live slightly
longer than the general U.S. White population.
For example, using mortality data based on records of the 1971 physician
population of 344,823 and the deaths of 19,086 physicians during the five year
period from 1969 – 1973, Goodman 6
calculated the remaining expectation of life of a male physician at age 45 years
to 30.7 years, which is 3 years more than the general U.S. White population.
For female physicians at age 45 years, it is 35.8 years of 2 years more
than that of the general U.S. White population.
With increasing age, this difference diminishes; at ages 80 years and
older , the life expectancies of physicians are only about one half of a year
longer. It should be noted,
however, that these data apply for physicians of all disciplines and
specialties. Of interest in this
context is the life expectancy of General Practitioners (G.P.’s), because
these would be the ones that would most likely be approached for counsel in
regards to achieving longevity. According
to Goodman 6
, G.P’s have the shortest lifespan of all medical specialties.
Their standard mortality rate below age 55 for the years 1969 – 1973
was 140.4%, at above 55 years, 111.4% above the average mortality of all
physicians. For specialists, the
standard mortality rates compared to all physicians were 89.1 and 80%, for ages
below and above 55 years, respectively. This
means that close to 60% more G.P’s die below the age of 55 years than
specialists, above 55 years of age, the difference amounts to 31%.
From this it can be calculated that the average expectation of life of
G.P.’s is about 5 years shorter than that of the US White population.
The shorter lifespan of G.P.’s was
attributed to the fact that they begin their careers at an earlier age and thus
are subjected to the stress of practice for longer period than specializing
physicians. That practicing
(general) physicians have a short life span became apparent as soon as
sufficiently reliable mortality data became available.
For example, an article on the longevity of physicians appearing 7
in 1896 specifically mentions the short life span of the members of the
profession and acknowledges long lived doctors as “…comforting
exceptions…”. Similarly, an
Editorial published in JAMA in 1902 admits that practicing physicians have the
shortest life span of all professions. 8
While stress undoubtedly is a major factor which shortens their lifespan,
neglect of their own health and unhealthful habits also contribute.
According to a survey published in 1984, no less than 59% of the
physicians questioned considered themselves overweight, 73% felt they were not
exercising enough, 24% admitted to frequent drinking and 15% were smokers. 9
The same report concludes that “there may be a far larger group of
subclinically impaired physicians whose health habits may not only affect their
own longevity but their practice of medicine as well,”
which in essence proved Wallach’s point.
There are, of course, physicians who reach long life spans by living
healthily or by making appropriate lifestyle changes when necessary.
A case in point is provided
by Dr.Benjamin Spock, who died at the age of 94 years.
According to newspaper reports, Spock at the age of 88 years was
suffering from a serious respiratory ailment that clogged his lungs.
He was put on antibiotics for 9 months, to no avail.
On insistence of his second wife, Morgan, Spock abandoned conventional
treatment in 1991, became a vegetarian and was put on a vegetarian died.
He lost 50 pounds in six weeks, his lungs cleared and he could stop
taking the antibiotics. Cultures with long-lived populations – do they exist? Wallach mentions 5 cultures whose populations attain
unusually long life spans. He is
drawing his knowledge from published and credible sources.
It thus must astonish when his critics argue that no such cultures exist.
The best know of these cultures are the Hunzas of Kashmir, about whom
numerous books have been written. There
can be no doubt that many of them live at least to 100 years of age, although
the actual ages claimed in individual cases may be somewhat uncertain.
Physicians such as Sir Robert McCarrison established that the Hnnzas
remain remarkably free of disease during their long lives.
This has been attributed to lifestyle, diet, agricultural practices and
the mineral rich glacier milk used for irrigation and as drinking water. 10
The villagers of Vilacabamba in Ecuador, similarly are famous for their
longevity and good health. According
to extensive clinical studies, this population rarely develops arteriosclerosis,
diabetes or solid tumors. Lymphocytic
leukemia was the only malignant condition found and was observed only in some
very old people toward the end of their lives.11
The health and long life spans of the Vilacabambans were attributed to an
apparently optimal mineral composition of the soil, plants and water in the
area.12 Other cultures with long life spans are the Russian
Georgians, the Abkhazians, Azerbaijanis and Armenians, the peoples residing near
Lake Titicaca and some tribes of Tibet. In
his book “Rare Earths: Forbidden Cures,” Wallach shows the copy of a May
1973 newspaper clipping showing the Azerbaijani
Shjimos Mislimov at his 168th birthday.
Mislimov, who died in September of the same year, 13
as he was at the time the oldest person living in the Soviet Union, a special
postage stamp was issued to honor him. Based on this evidence it would thus seem difficult to deny
the existence of long lived cultures. Long
life spans are also increasingly reached in the Western industrialized nations.
In the early 1960’s, centenarians were still quite rare; in Hungary
(population: 10.3 million), 23 were found in 1961, in Austria (population 7.4
million), 17 (cf. “Geriatric,” L. Heilmeyer et al. eds., Thieme Verl.
Stuttgart, 1966, p. 228). In 1966,
only 4000 People living in the U.S.A. were around one hundred years old.
Today the number is estimated to be 40,000.
The number is likely to increase during the next decades, and along with
it more and more people will reach even longer life spans.
According to Wallach, the biological life span of humans is about 120 –
140 years. However, the upper limit
of the human life span is actually unknown.
The example of Mislimov proves that one can life longer than that, and
Wallach mentions the Chinese doctor Li, who allegedly died at the age of 256
years. While skeptics tend to be
critical of claims of extreme longevity, the fact is that well documented
longevity records are now broken with increasing frequency.
Thus, during the next decades, we may expect a further increase of the
number of “supracentenarians.” This
could be in part because, for the first time in human history, people will reach
the age of over 100 years who were born after the discovery of vitamins.
Don’t let us forget that vitamin C is known only since 1910, and most
other vitamins were discovered even later than that.
In the developed countries, people are now growing up of whose parents
were able to take advantage of the new nutritional and medical discoveries prior
to their birth and during their entire life span. Dr. Wallach’s Cystic Fibrosis research. Wallach’s contributions to cystic fibrosis (CF) research
and therapy were rejected in 1979, and even today his American critics declare
his findings as invalid, even though other researchers are now discussing the
disease in quite similar terms. At
the very least, Wallach deserves recognition for being the first to observe
pancreatic lesions typical of CF in 3 young rhesus monkeys in the obvious
absence of a genetic defect. The monkeys were born in Yerkes Primate Research
Center 14
by mother animals that had been given extra doses of vegetable oil to
treat a skin condition during pregnancy. Wallach proposed on the basis of these findings that CF is
an environmentally induced disease and suggested the CF could be prevented and
treated by selenium and other antioxidants.
He conducted a survey of 120 families with one or more CF children and
concluded that the history and patient profile was consistent with an acquired
environmental disease caused by a perinatal deficiency of selenium, zinc and
riboflavin, and CF can be exacerbated by diets which are also low in vitamin E
and rich in polyunsaturated fatty acids. His work triggered the interest of a group of physicians
who were looking for ways to help their CF patients.
A few years later these doctors were so impressed with Wallach’s
therapy that they awarded him the “Wooster Beach Gold Medal” in 1988 and
nominated him for a Nobel Prize in 1991. This
nomination did not follow the prescribed procedure and was not recognized by the
Nobel Prize Committee. Critics now
hold the mentioning of this nomination against
him as any such nomination should be kept strictly confidential.
While this is true., it is actually a very minor point which should not
be used to detract from his contribution. To demonstrate the CF like pancreatic lesions develop in
populations residing in regions naturally low in selenium, Wallach and his wife,
Ma Lan, M.D., a Chinese physician, traveled to China to conduct more research.
In collaboration with researchers at Harbin Medical University, Wallach
showed that hitherto ignored pancreatic lesions occurred in 35% of 1700
documented cases of Keshan disease (KSD), which is a cardiomyopathy occurring in
low selenium regions of China. 15
In the meantime, other researchers have drawn attention to the aberrant
oxygen free radical activity and the low selenium and antioxidant status in
cystic fibrosis patients. 16
17
18
19 Although perinatal selenium and antioxidant vitamin
deficiency is still not accepted as a cause of CF, it is agreed that selenium
deficiency my develop in CF children because of digestive malabsorption or after
prolonged total parinateral nutrition. 20
21
22
A case of cardiomyopathy in a CF patient which was caused by selenium
deficiency has also been described.23
The therapy of CF patients with selenium and antioxidant vitamins has
since also been tested in a clinical trial.
One German group 24
concluded: “In cystic fibrosis
(CF) patients the antioxidative balance is chronically disturbed.
Free radicals were generated by bronchial-pulmonal infection and
additionally (there) exists a deficiency of antioxidative substances by enteral
malabsorption especially (of) vitamin E and selenium…For CF patients therefore
we recommend a sodium selenite substitution therapy, best in combination with
vitamin E.” Amalgam fillings and multiple sclerosis. Dr. Wallach mentions mercury from dental amalgam fillings
as a cause of multiple sclerosis (MS). His
critics argue that this is not proven, which is true, but authorities on MS
would also agree that mercury is definitely on the list of suspect causative
agents. The hypothesis that MS is
caused by an allergic reaction to mercury from dental amalgam was first proposed
in 1966 by Ernst Baasch. 25
This neurologist at University of Zurich became interested in the effects
of mercury after he himself developed MS following the insertion of dental
amalgam fillings. 26
In his subsequent study he noted the presence of amalgam fillings in 498
of 500 consecutively examined MS patients.
As amalgam fillings are so common in the general population, he cautioned
that this does not prove nor disprove a causal relationship.
Baasch further found that 2 MS patients improved after they had their
amalgam fillings removed. Another
patient reportedly developed MS after she received her first amalgam fillings at
19 years of age; this patient, according to Baasch, could have been sensitized
to mercury because she had been treated with mercury for congenital syphilis at
8 years of age. 27
Finally, this author also suggested that other toxic metals could be
factors in the causation of MS. Studies
of the mercury/amalgam status of 100 MS patients revealed that 11 of these
patients had previously been treated with mercury ointments.
The acute exacerbation of MS symptoms during removal (pulverization) of
one old filling was reported by Ingalis. 28
Because of the apparent connection of mercury exposure with MS and other
neurodegenerative diseases. 29 Cardiomyopathy – a selenium deficiency disease? Wallach states that cardiomyopathy is caused by a selenium
deficiency, his critics counter that cardiomyopathy is really an entire group of
heart muscle diseases with several different causes.
Wallach uses a pathologically more precise definition of cardiomyopathy,
based on the detection of oxygen radical damage of the heart muscle.
Oxygen radical damage of the myocardium can occur in many diseases.
Since selenium prevents the generation of oxygen radicals, cardiomyopathy
is caused by primary selenium deficiency in regions naturally low in selenium,
as was first shown to be the case in the Keshan Disease regions of China. 30
Cardiomyopathies due to selenium deficiency were at first considered
unlikely to develop in the Western industrialized nations until they were shown
to occur in patients after prolonged total parenteral nutrition, in subjects
with destructive lifestyles such as alcoholics, in patients suffering from
intestinal malabsorption or from diseases resulting in decreased Se retention
such as AIDS and in cancer patients treated with certain catatonic drugs.
Selenium deficiency also plays a role in the causation of Coxsackie
B-virus (CBV) induced cardiomyopathies, as nonpathogenic strains of CBV have
been shown to become highly pathogenic under conditions of selenium deficiency.
31
Selenium deficiency thus is the major cause of cardiomyopathy.
Classical textbooks of cardiology, even the newest excellent and up to
date reference book on “Nutritional Influences on Illness” [2nd
Edition 1993, Third Line Press, Tarzana, Calif., p. 189], clearly states that
selenium deficiency is associated with the development of cardiomyopathy, while
deficiencies of other agents, e.g. magnesium, L-carnitine, coenzyme Q only may
be factors in the development of the condition. Copper deficiency – a cause of aneurysms, graying hair
and facial wrinkles? Wallach is being criticized for stating that all aneurysms
are caused by a copper deficiency, when he in fact only claims that aneurysms
are most frequently caused by copper deficiency.
That this is true is know from studies in many animal species (e.g. pigs,
guinea pigs, rabbits, cattle, chicks, turkeys, etc.) Copper is needed for elastin synthesis, specifically for
the oxidative deamination of lysine. Diminished
deamination of this amino acid causes less lysine to be converted to desmosine,
the cross linking group of elastin. This
results in fewer cross linkages in this protein, which, in return, results in
less elasticity of the aorta. 32
Copper deficiency in humans was considered rare in humans but is now
becoming a concern primarily in pregnancy.
In a recent study with 20 pregnant women on self selected diets, positive
balance was observed only if a copper supplement was consumed.
33 Copper deficiency need not be caused solely by low dietary
copper intakes; copper deficiency may be induced by dietary components, notably
fructose and ascorbic acid; some also consider excessive zinc as a possible risk
factor. In all, rather than being
criticized, Wallach should be given credit for drawing attention to the
important role of copper deficiency in the pathogenesis of aneurysms. The fact that copper influences the pigmentation of hair is
well supported by observations with copper deficient animals.
Experiments conducted in the early 1930’s showed that the fur of black
coated rats turned gray when they were placed on a copper deficient diet.
Achromotrichia has been described in other species deficient in copper:
rabbits, dogs and sheep. Copper is
known to be required for the transformation of tyrosine to melanin.
In copper deficiency, the physical nature of hair is also affected, it
becomes brittle and crinkled because oxidative processes which give hair its
normal elasticity require copper. 34
Other factors contribute to the graying of hair, a deficiency of
pantothenic acid, for example. Clinical
studies of the effects of copper supplementation on hair color in humans are
lacking but Wallach does report one case in which gray hair regained
pigmentation in a woman after supplementing with copper.
Wince copper is required for elastin and collagen biosynthesis, changes
of elastic connective tissues are expected to occur in copper deficiency.
Since 75% of the typical diets in the United States furnish less than the
current daily requirement of 2mg of copper per day, 35
chronic copper deficiency thus could indeed contribute to hair depigmentation
and skin wrinkling, especially in women. In
a recent study with 20 pregnant women on self selected diets, positive balance
was observed only if a copper supplement was consumed. 36 Alzheimer’s disease – does it occur in pigs and can
it be cured? Wallach was also criticized for suggesting that 50% of 70
year old Americans have Alzheimer’s disease and also because he claims to have
cured pigs with “Alzheimer’s” disease, when pigs are not known to develop
this disease. As to the first
point, Dr. Wallach said “one out of two people who reach the age of 70 years gets
the disease,” he did not say
“one out of two people who reach the age of 70 years has the
disease”! As to his claim of having cured Alzheimer’s disease in
pigs, he was referring to a condition which develops in pigs when fed a diet
high in polyunsaturated fat and low in selenium and vitamin E.
This condition pathologically resembles Alzheimer’s disease and can be
cured with vitamin E. Oxidative
stress is increasingly recognized to play an important role in the pathogenesis
of Alzheimer’s disease, and many researchers now believe that Alzheimer’s
disease should be preventable by supplementing with appropriate antioxidants. Malabsorption disease:
It does exist! While Wallach claims that many Americans suffer from
‘malabsorption disease,’ his critics argue that this is a nonexistent
disease, like the long discredited idea of autointoxication.
Here, again, one must side with Wallach, since malabsorption disease,
a.k.a. malabsorption syndrome, celiac disease, sprue syndrome, nontropical sprue,
idiopathic steatorrhea is a well characterized and relatively common disease. 37
38
In his book, “Let’s Play Doctor,” Wallach provides a correct
description of celiac disease and its treatment in lay terms.
Nowhere in his books or lectures does Wallach mention autointoxication, a
condition which was widely diagnosed in the early 20th Century 39
but which is no longer recognized as a defined disease entity. Male pattern baldness and dietary tin – unproven
hypothesis? In his lectures, Wallach occasionally mentions that male
patter baldness is caused by tin deficiency.
The basis for this claim is that he observed significant hair regrowth on
himself following tin supplementation. Male
pattern hair loss was originally reported by Klaus Schwarz et. al.
40
to develop in tin deficient rats, and these findings were subsequently confirmed
in a 1990 study by Yokoi et al 41
of Kyoto University. The claimed
stimulation of hair growth by tin at high dilutions thus is not an unfounded
idea although it is not clear whether the hair regrowth he observed in his self
experiment was actually or solely due to the tin present in the plant derived
mineral extract he was ingesting Diabetes, chromium and vanadium. Wallach’s claim that diabetes and hypoglycemia are due to
vanadium and chromium deficiencies was stated to be unsupported by clinical
research. There is, however,
abundant published evidence indicating a role of chromium and vanadium ion the
insulin system; see papers Ref. 42
–44, and references cited therein. According
to USDA’s Richard Anderson, 42
suboptimal intakes of chromium by people consuming average diets may lead to
signs and symptoms of chromium deficiency that include elevated blood glucose,
insulin, cholesterol and triglyceride concentration and decreased insulin
binding and receptor number. Extreme
signs of Cr. Deficiency were observed in TPN patients and were corrected by Cr
supplementation. Recent clinical
trials with vanadium have also yielded positive effects on the glucose/insulin
system. 43 Sodium consumption and high blood pressure. Wallach’s critics sometimes single out statements or
opinions which he made or supposedly has made which appear to identify him as
uninformed. One of such statements
is that sodium consumption is unrelated to high blood pressure in humans.
In a recently published review on the role of dietary salt in
hypertension it is stated, 44
“Most people can eat as much NaCl as they like…and nothing happens to blood
pressure.” That a few, especially
those with kidney disease, do not excrete it as fast as it is taken in and
respond with a rise of blood pressure, is rather generally known and also known
to Dr. Wallach. Low back pain and osteoporosis. Wallach was accused of expressing the “absurd idea”
that all low back pain is due to osteoporosis.
However, Wallach only mentions osteoporosis as a contributing cause of
low back pain. He is (correctly)
linking calcium and copper deficiency with the initiation of osteoporosis which
he then claims triggers disk degeneration and back pain.
In his book “Let’s Play Doctor,” he writes: “Bachache is usually
a muscle strain from overwork and/or a subluxation resulting from a fall, auto
accident of improper lifting technique. On
occasion, a serious case of constipation will cause a ‘backache’ from
impacted stool or pressure from gas…Prevention includes proper lifting
technique, strengthening exercises, proper nutrition including calcium (2000 mg)
and magnesium (800 mg), high fiber diets and eight glasses of water per day.” Are periodontal disease and Bell’s palsy caused by
calcium deficiency? Wallach’s critics may find fault with his claim that
calcium deficiency may cause periodontal disease, but in “Nutritional
Influences on Illness,” by M.R. Werbach (Third Line Press, Tarzana, Calif.), 2nd
Ed., 1993, p 672, periodontal disease is specifically associated with calcium
deficiency. Calcium deficiency
is a logical cause of periodontal disease since it promotes bone loss.
As calcium deficiency is widespread in the general population, Wallach
deserves credit for reminding us of the importance of calcium in this context.
In his book, “Let’s Play Doctor” he makes a good case for the
treatment of periodontal disease with supplemental calcium, magnesium, zinc,
etc. So far as Bell’s Palsy is
concerned, he correctly states in the same book that Bells Palsy is caused by an
inflammation, swelling or squeezing of the facial nerve.
He recommends a treatment
which involves not only the administration of calcium, but also of magnesium,
essential fatty acids, American ginseng, colloidal minerals and vitamin B12. Concerning the so called “Colloidal Minerals.” “Colloidal minerals” comprise a group of liquid mineral
supplements which are produced by leaching deposits of humic shales with water.
These extracts are claimed to contain mineral in highly bioavailable
forms and have other unusual properties, all of which ware disputed by the
critics, primarily because colloidal substances, in general, are not absorbed.
In this case the argument rests on the question of nomenclature.
The extracts were originally thought to contain the minerals
predominantly in colloidal forms. It
is now know that they contain the minerals in ionic as well as in colloidal
forms. Because the term
“colloidal” may give rise to misunderstandings, the products are now
referred to as “liquid” or “plant derived minerals.”
The first of these products has been marketed for more than 70 years and
was claimed by the original promoters to have been used as a remedy by local
native Americans, which is not as far fetched as it would seem since these had
extensive knowledge of healing plants and minerals.
It has been claimed that these products my be contaminated by radioactive
elements or contain organic compounds that could be carcinogenic, estrogenic or
stimulate the immune system, etc., but none of this is backed b evidence.
As to the superior bioavailability of liquid minerals as compared to
minerals in their elemental state, this claim is correct inasmuch as iron, which
was widely used in the elemental form for supplementation has a very low
bioavailability and liquid iron salts are know to be well absorbed.
A more detailed discussion of liquid minerals may be found elsewhere. 46 Dr. Wallach’s publications. A partial list of Dr. Wallach’s publications, reviews and
books authored or coauthored during the period from 1965 to 1994 comprises 55
titles: 20 of his papers were published in the Journal of the American
Veterinary Medical Association (JAVMA); 6 reviews appeared in professional
books, the remainder in other professional veterinary journals.
Wallach is also the coauthor (with W.J. Boever) of an authoritative
treatise, “Diseases of Exotic Animals: Medical and Surgical Management”
published by W.B. Saunders Co., Philadelphia in 1983. Wallach’s research papers reflect his wide range of
interests and experience. His first
paper appearing in 1965 describes goitrogenic hypothyroidism in feeder lambs;
subsequent articles and reviews deal with common diseases and treatments for
waterfowl, game birds, exotic birds, reptiles, fish ruminants, kangaroos,
monkeys, elephants, nutritional problems of captive exotic animals, descriptions
of a case of degenerative arthritis in a black rhinoceros, of visceral gout and
nutritional problems in captive reptiles, angioedema in a gorilla, fibrous
osteodystrophy and hypervitaminosis D in green iguanas, the immobilization of
small and very large animals (rabbits, Guinea pigs, African elephants),
steatites in captive crocodiles, the anaesthesia of reptiles, the hand rearing
of a white rhinoceros, surgical techniques for caged birds, the foot care for
captive elephants, erysipelas and cystic fibrosis. Summary and concluding remarks. As a veterinarian and pathologist working in zoos, Dr. Joel D. Wallach had the unique opportunity to observe, diagnose, treat and autopsy a great variety of exotic animals. This led him to recognize the importance of nutrition and especially minerals in health and disease. His discovery, in 1979, of cystic fibrosis like pancreatic lesions in rhesus monkeys and their nutritional causes led him to propose an alternative etiological hypothesis of the disease in humans and make new treatment recommendations involving antioxidant vitamins and trace elements. Although his ideas were rejected at the time, they are now being rediscovered by others. After years of practice as a naturopathic physician, Wallach entered the multilevel marketing business and became nationally know as the author of “Dead Doctors Don’t Lie.” In this lecture, Wallach voices his opinions on numerous medical and scientific issues. Wallach’s popularity and success has led some of his detractors to question his credentials and the veracity of some of his statements. The present account shows that Dr. Wallach’s academic record is unassailable, and that his opinions and views are generally well substantiated. If he startles some of his critics this may be because developments in his area of expertise are not generally know or ignored by the largely drug oriented conventional medicine. [1] G.N. Schrauzer, Ph.D. , Professor emeritus, Address for correspondence: Biological Trace Element Research Institute and Information Center, 11526 Sorrento Valley Rd., Ste. A. San Diego, CA, 92121 1 [1] J. Pontolillo, “Colloidal Mineral Supplements: Unnecessary and Potentially Hazardous,”p.1; ),www.quackwatch.com/01QuackeryRelatedTopics/DSH/colloidalminerals. (National Council of Health Fraud) Newsletter 19 (2) March-April 1996 Issue. 2 [2] E.J. Thacker and K.C. Beeson (1958): Occurrences of mineral deficiencies and toxicities in the United States and problems of their detection. Soil Sci. 85ii 87-94. 3 [3] J. Kubota and W.H. Allaway. D.L. Carter, E.E. Cary and V.A. Lazar. 1967. Selenium in relation to soils and forage plants of the United States in relation to selenium responsive diseases of animals. Agric. Food Chem. 15: 562 – 565. 4 [4] L.C. Clark et al. The Nutritional Prevention of Cancer with Selenium 1983 – 1993. JAMA 276: 1957 – 1963. 5 [5] J.D. Beasley and J.J. Swift (1989): The Kellogg Report. The impact of Nutrition, Environment and Lifestyle on the Health of Americans. The Institute of Health Policy and Practice, The Bard College Center, Annondale-on-Hudson, New York, 12502, Library of Congress Catalog Card Number: 89-84263, p. 169. 6 [6] L.J. Goodman (1975) Longevity and mortality of American Physicians, 1969-1973. Milbank Memorial Fund Quarterly; Health and Society 53(3): 353 –375. 7 [7] Editorial, JAMA, 1902, issue of Oct. 25, p. 1053 –4. 8 [8] G.M. Gould and W.L. Pyle,. Anomalies and curiosities of medicine. The Julian Press 1896, p. 380-381. 9 [9] K.B. Wells, C.E. Lewis, B. Leake, J.E. Ware, Jr. (1984). Do Physicians Preach what they Practice?” JAMA 252: 2846 – 2848. 10 [10[ J.I. Rodale (1949), “The Healthy Hunzas.” Rodale Press, Emmaus, PA, 1949. 11 [11] F.Silio, A. H. Laguna, L.e. Garcia, J.G. Guaman, M.S. Salvador (1966) Mineral composition of foods and drinking water from Vilacabamba. In: Proc. Metal Ions in Biology and Medicine Vol. 4; Ph. Collery, J. Corbello, J.L. Domingo, J. D. Etienne, J.m. Llobet eds. John Libbey Eurotext, Paris, 1996, pp 563-565. 12 [12] a: M. Salvador (1972) Vilacabamba, Tierra de Longevos. Caa de la Cultura Ecuatoriana, Quito, 1972 b: M. Salvador (1980) : Aterosclerosis en Vilacabamba. Medicina y Ciencias biologicas XVI, 28. 13 [13] M. Williams-Sarkisian, B. Apisson: A diet for happy, healthy 100 years,” German Edition, Die Kaukasus-Diat, Ullstein Verlag Frankfurt, 1985. 14 [14] J.D. Wallach, B. Garmaise (1979): Cystic fibrosis – A perinatal manifestation of selenium deficiency. In: Hemphill D.D. ed., Trace Substances in Environmental Health XIII, pp. 469 – 476. 15 [15] J.D. Wallach, Ma Lan, Wei Han Yu, Bo-Qi Gu, Feng Teng Yu and Roy F. Goddard (1990). Common denominators in the etiology and pathology of visceral lesions of cystic fibrosis and Keshan Disease. Biol. Trace El. Res. 24: 189 – 205. 16 [16] P. Foucand, P. Therond, M. Marchand, F. Brion, F.F. Demelier, J. Navarro (1988). Selenium et vitamin E au cours de la mucoviscidose. Arch. Fr. Pediatr. 45(6), 383 –6. 17 [17] A.G. Thoman, V. Miller, A. Shenkin, G.S. Fell, F. Taylor (1994). Selenium and glutathione peroxidase status in pediatric health and gastrointestinal disease. J. Pediatr Gastroent Nutr 19, 2: 213 – 219. 18 [18] B. Salh, K. Webb, P.M. Guyan, J.P. Day, D. Wickens, J. Griffin, J.M. Braganza, T.L. Dormand (1989), Clin. Chem. Acta 181 (1) 65-74. 19 [19] B.M. Winklhofer-Roob (1994): Oxygen free radicals and antioxidants in cystic fibrosis: the concept of an oxidant- antioxidant imbalance. Acta. Paeditr Suppl. 395: 49 – 57. 20 [20] R.D. Watson, R.A. Cannon, G.S. Kurland, K.L. Cox, F.C. Frates (1985), Selenium responsive myositis during prolonged home total parenteral nutrition in cystic fibrosis. JPEN J Parenteral Enteral Ntr. 9(1) 58 – 60. 21 [21] C. Dominguez, M. Llovera, E. Ruiz, V. Araujo, S. Linan, S. Gartner, N. Cobos, “Antioxidant trace elements, glutathione and glutathione peroxidase in cystic fibrosis patients: relation to lipid peroxidation status. In: Proc. Conf. Metal Ions in Biology and Medicine, Vol. 4; Ph. Collery, J. Corbello, J.L. Domingo, J.D. Etienne, J.M. Llobet eds., John Libbey Erotext, Paris, 1996, pp. 592 – 595. 22 [22] B. Dworkin, L.J. Newman, S. Berezin, W.S. Rosenthal, S.M. Schwarz, L. Leiss (1987). Low blood selenium levels in patients with cystic fibrosis compared to controls and healthy adults. JPEN J. Parenteral and Enteral Nutr. 11(1) , 38 – 41. 23 [23] D.M. Volk, S.A. Cutliff (1986), Selenium deficiency and cardiomyopathy in a patient withcystic fibrosis. J.Ky. Med. Assoc. 84 (5) 222-4. 24 [24] E. Kauf, E. Janitzky, L. Vogt, K. Winnefeld, H. Dawczynski, M. Forberger, G. Jahreis, H. Vogel (1995). “The significance of a selenotherapy in cystic fibrosis patients” Med. Klin. 90, Suppl. I, 41-45. 25 [25] E. Baasch (1966), Theoretische Uberlegungen zur Atiologie der Scierosis multiplex. Die Multiple Sklerose eine Quecksilberallergie? Schweizer Archiv f. Neurologie, Neurochirurgie und Psychiatrie 98 (1) 1-19. 26 [26] E. Baasch, personal communication to author, 1995. 27 [27] Knolle and Gunther G. Knolle and B. Gunther (1967). Beitrag zur Atiologiehypothese: Amalgam und Multiple Sklerose. Schw. Monatsschr. Zahnheilk. 77. 761- 776. 28 [28] T.H. Ingalis (1986): Triggers for multiple sclerosis. Lancet 2 (1986) 160. 29 [29] H. Visser (1995): Indikationed und Kontraindikationen der Amalgamfullung. In: Status quo and perspectives of amalgam and other dental materials. L.T. Friberg and G.N. Schrauzer, eds. G. Thieme Verlag Stuttgart, New York, p.35. 30 [30] G.Q. Yang (1985), Keshan disease: an endemic selenium related deficiency disease. In: Trace Elements in Nutritional and Children. R.K. Chandra, ed. Raven, New York, pp. 273-290. 31 [31] O.A. Levander and M.A. Beck (1997). Insights from Coxsackie B Virus induced myocarditis in mice deficient in selenium and vitamin E. Biol. Trace El. Res. 56 (1) 5 – 21. 32 [32] H.L. Keil and V.E. Nelson (1931). The role of copper in hemoglobin regeneration and reproduction. J. Biol. Chem. 93: 49 33 [33] E.J. Underwood: Trace Elements in Human and Animal Nutrition., 3rd Ed., Academic Press, New York and London, 1971, pp 53 – 115. 34 [34] D.M. Danks, B.J. Stevens, P.E. Campbell, J.M. Gillespie, J. Walker-Smith, J. Blomfield and B. Turner (1972), Menke’s kinky hair syndrome. Lancet, 1: 1100 – 1102. 35 [35] L.M. Klevay and D.M. Medeiros (1966), Deliberations and Evaluations of the Approaches, Endpoints and Paradigms for dietary recommendations about copper. J.Nutr. 126: 2419S – 2419S. 36 [36] Taper L.J., et al. (1981), Zinc and copper retention in pregnant women. Fed. Proc. 40: 855. 37 [37] Current diagnosis and treatment, MA. Krupp, M.J. Chatton, S. Margen (1971), eds., Lange Medical Publications, Los Gatos, Calif., pp. 329 – 330, and references cited therein. 38 [38] N.J. Greenberger and K. Isselbacher; Disorders of Absorption. In: Harrisons Principles of Internal Medicine, 6th Edition, MacGraw-Hill Book Company, New York, pp. 1467 – 1484. 39 [39] J.H. Kellogg: Autointoxication or Intestinal Toxemia. 2nd Ed., The Modern Medicine publishing Co., Battle Creek, Michigan, 1922. 40 [40] K. Schwarz, D.B. Milne, and E. Vinyard (1970) Growth effect of tin compounds in rats maintained in a trace element controlled environment. Biochim Biophys Res. Comm. 40, 22-29. 41 [41] K. Yokoi, M. Kimura and Y. Itokawa (1990), Effect of dietary tin deficiency on growth and mineral status in rats. Biol Trace E. Res. 24: 223. 42 [42] R.A. Anderson (1997): “Nutritional factors influencing the glucose/insulin system: Chromium. J. Am. Coll. Clin. Nutr. 16: 404 – 410. 43 [43] S. Verma, M.C. Cam and J.H. McNeill (1998). Nutritionaly factors that can favorably influence the glucose/insulin system: Vanadium. J.Am. Coll Nutr. 17 (1) 11-18. 44 [44] H.G. Preuss., T. Jarrell, R. Scheckenbach, S. Lieberman, R.A. Anderson (1998). Comparative effects of chromium, vanadium and Gymnma on sugar induced blood pressure elevations in SHR. J. Amer. Coll. Nutr. 17 (21) 116 –123. 46 [46] G.N. Schrauzer (1999), An evaluation of liquid vitamin and mineral technology. J. of Medicinal Foods, in press. *Whenever the term "colloidal minerals" is used in association with Dr. Joel Wallach or his famous audio tape Dead Doctors Don't Lie, it is important to note that Dr. Joel Wallach is referring to organic colloidal minerals. Organic colloidal minerals are minerals that have been processed through a plant, a fact which greatly increases the rate at which the human body can absorb these minerals. Dr. Joel Wallach is NOT referring to colloidal minerals mined from inorganic sources such as seabeds or clays. Many of Americas largest colloidal minerals companies sell inorganic colloidal minerals. The statements and / or products found on this website have not been evaluated by the Food and Drug Administration. Products mentioned or appearing on this website are not intended to diagnose, treat, or prevent any disease or disease condition. American Longevity Independent Associate, ID # 6570401
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